1. Coronavirus: could it be burning out after 20% of a population is infected?  The Conversation UK
  2. Coronavirus may have reached Europe as early as March 2019, study claims  Mirror Online
  3. Existing drugs can prevent SARS-CoV-2 from hijacking cells  Medical Xpress
  4. Was coronavirus really in Europe in March 2019?  The Conversation UK
  5. Scientists ID drugs that may block coronavirus from jumping to uninfected cells  The Financial Express
  6. View Full coverage on Google News
There is speculation about whether a population can achieve some sort of immunity to the virus with as little as 20% infected.There is speculation about whether a population can achieve some sort of immunity to the virus with as little as 20% infected.

Coronavirus: could it be burning out after 20% of a population is infected?

Coronavirus: could it be burning out after 20% of a population is infected?

They said sometimes this hijacking interferes with the activity of the host's proteins and other important molecules such as enzymes, which change protein activity by making chemical modifications to its structure.They said sometimes this hijacking interferes with the activity of the host's proteins and other important molecules such as enzymes, which change protein activity by making chemical modifications to its structure.

Scientists identify drugs that may block coronavirus from jumping to uninfected cells - The Economic Times

How exactly does the antiviral drug remdesivir counter SARS-CoV-2 – the coronavirus strain responsible for COVID-19? And how well?How exactly does the antiviral drug remdesivir counter SARS-CoV-2 – the coronavirus strain responsible for COVID-19? And how well?

How remdesivir works, and why it's not the ultimate coronavirus killer - Scope

Research explores the interaction between airway and immune cells to determine why some people are at greater risk than others of severe COVID-19.Research explores the interaction between airway and immune cells to determine why some people are at greater risk than others of severe COVID-19.

Why are some people at greater risk of severe COVID-19?

People living with HIV are learning how to cope with a new pandemic.People living with HIV are learning how to cope with a new pandemic.

COVID-19 and HIV: What You Need to Know - POZ

Kinases are cellular control switches. When they malfunction, they can cause cancer. The coronavirus hijacks these kinases to replicate, and cancer drugs that target them could fight COVID-19.Kinases are cellular control switches. When they malfunction, they can cause cancer. The coronavirus hijacks these kinases to replicate, and cancer drugs that target them could fight COVID-19.

Coronavirus and cancer hijack the same parts in human cells to spread – and our team identified existing cancer drugs that could fight COVID-19

Coronavirus and cancer hijack the same parts in human cells to spread – and our team identified existing cancer drugs that could fight COVID-19

 

GROWING OLD UNGRACEFULLY: All Creatures Great and Small | Lost Coast Outpost | Humboldt County News

The researchers note that these findings are in line with a recent study, published in Cell, that showed a robust T cell response in individuals with moderate cases of Covid-19.The researchers note that these findings are in line with a recent study, published in Cell, that showed a robust T cell response in individuals with moderate cases of Covid-19.

New Research: Lessons from immune response of most severe Covid patients | Explained News,The Indian Express

Coronavirus transforms the infected cells in a highly unusual manner, forcing them to grow long, protruding branches, which penetrate multiple healthy cells at once, a new study has found.Coronavirus transforms the infected cells in a highly unusual manner, forcing them to grow long, protruding branches, which penetrate multiple healthy cells at once, a new study has found.

Coronavirus makes infected cells sprout ‘sinister’ tentacles to quickly travel inside the body, new study claims — RT World News

Researchers found that SARS-CoV-2 hijacks tendrils that grow from infected cells and may ride them to infect others. But existing compounds might slow their roll.Researchers found that SARS-CoV-2 hijacks tendrils that grow from infected cells and may ride them to infect others. But existing compounds might slow their roll.

Old Drugs Could Reveal a New Way to Attack the Coronavirus | WIRED

In the study, published on Sunday in the journal Cell, the scientists analysed all host and viral proteins that showed changes in an enzymatic process called phosphorylation after SARS-CoV-2 infection. In the study, published on Sunday in the journal Cell, the scientists analysed all host and viral proteins that showed changes in an enzymatic process called phosphorylation after SARS-CoV-2 infection.

Scientists Identifies Drugs that May Block Coronavirus from Jumping to Uninfected Cells

Most antivirals in use today target parts of an invading virus itself. Unfortunately, SARS-CoV-2 – the virus that causes COVID-19 – has proven hard to kill. But viruses rely on cellular mechanisms in human cells to help them spread, so it should be possible to change an aspect of a person’s body to prevent that […]

Coronavirus and cancer hijack the same parts in human cells to spread – and some cancer drugs could fight COVID-19 – Raw Story